Felice B Aull, Ph.D.

Telling and listening: Constraints and opportunities
Aull, F
2005 OCT ;13(3):281-293, Narrative (Society for the Study of Narrative Literature)
— id: 58814, year: 2005, vol: 13, page: 281, stat: Journal Article,

Poetry in the borderlands of medicine
Aull, Felice
2005 Nov-Dec;37(10):698-700, Family medicine
— id: 61851, year: 2005, vol: 37, page: 698, stat: Journal Article,

What can poetry teach physicians?
Aull, Felice B
2005 Nov;118(11):1310-1310, American journal of medicine
— id: 93964, year: 2005, vol: 118, page: 1310, stat: Journal Article,

Poetry in the borderland of medicine: an elective course
Aull, Felice
2004;:- [Web site] Vital Lines/Vital Signs: a conference on poetry & medicine, April 23-25, 2004 Duke University,
— id: 150906, year: 2004, vol: , page: , stat: Web Site,

Medical intellectuals: resisting medical orientalism
Aull, Felice; Lewis, Bradley
2004 Summer;25(2):87-108, Journal of medical humanities
In this paper, we propose analogies between medical discourse and Edward Said's 'Orientalism.' Medical discourse, like Orientalism, tends to favor institutional interests and can be similarly dehumanizing in its reductionism, textual representations, and construction of its subjects. To resist Orientalism, Said recommends that critics--'intellectuals'--adopt the perspective of exile. We apply Said's paradigm of intellectual-as-exile to better understand the work of key physician-authors who cross personal and professional boundaries, who engage with patients in mutually therapeutic relationships, and who take on the public responsibility of representation and advocacy. We call these physician-authors 'medical intellectuals' and encourage others to follow in their path
— id: 43622, year: 2004, vol: 25, page: 87, stat: Journal Article,

Ethics and the humanities: "Death of the good doctor: lessons from the heart of the AIDS epidemic"
Aull F
2003 ;10(3p?-?):- 3, Medical ethics journal (Lahey Clinic)
— id: 43623, year: 2003, vol: 10, page: , stat: Journal Article,

Editors' choices from the Literature, arts, and medicine database: 10 year anniversary
Aull, Felice
New York : New York University, 2003,
— id: 771, year: 2003, vol: , page: , stat: ,

Commentary [on the Literature, Arts, and Medicine Database]
Aull, Felice
2003 Oct;78(10):1055-1055, Academic medicine
— id: 39037, year: 2003, vol: 78, page: 1055, stat: Journal Article,

Medical humanities at New York University School of Medicine: an array of rich programs in diverse settings
Krackov, Sharon K; Levin, Richard I; Catanese, Veronica; Rey, Mariano; Aull, Felice; Blagev, Denitza; Dreyer, Benard; Grieco, Anthony J; Hebert, Cristy; Kalet, Adina; Lipkin, Mack Jr; Lowenstein, Jerome; Ofri, Danielle; Stevens, David
2003 Oct;78(10):977-982, Academic medicine
The New York University School of Medicine has a rich tradition of cultivating programs in medical humanities and professionalism. They are drawn from the departments, centers, students, and faculty in the School of Medicine, have linkages throughout the university, and are interwoven into the fabric and culture of the institution. Some are centrally based in the School of Medicine's deans' office, and others are located in individual departments and receive support from the dean's office. This article describes representative programs for medical students and faculty. Curricular initiatives, the fundamental components of medical students' learning, include a course entitled 'The Physician, Patient, and Society,' a clerkship essay in the Medicine Clerkship, an opportunity for reflection during the medicine clerkship, and a medical humanities elective. In 2002, the Professionalism Initiative was launched to enhance and reflect the values of the medical profession. Its curriculum consists of a series of events that coordinate, particularly, with existing elements of the first-year curriculum (e.g., orientation week, a session during anatomy, a self-assessment workshop, and a peer-assessment workshop). The Master Scholars Program is a group of five, theme-based master societies consisting of faculty and students who share common interests around the society's themes. Programs developed for the societies include colloquia, faculty-led seminars, a mandatory student-mentoring program, and visiting scholars. Finally, the authors describe three high-quality literary publications created at New York University School of Medicine. Each of the initiatives undergoes regular critical examination and reflection that drive future planning
— id: 39038, year: 2003, vol: 78, page: 977, stat: Journal Article,

Emigration
Aull F
2000 Nov;75(11):1114-1114, Academic medicine
— id: 111701, year: 2000, vol: 75, page: 1114, stat: Journal Article,

Resources for teaching literature and medicine
Aull F
Teaching literature and medicine New York : Modern Language Association, 2000,
— id: 2606, year: 2000, vol: , page: 368, stat: Chapter,

Commentary on "Ma, a memoir"
Aull F
1998 ;73(1):66-67, Academic medicine
Presents a short story entitled Ma, A Memoir (L. Freed, 1996) to illustrate some of the characteristics of dementia to aid medical students and other inexperienced health care workers in distinguishing a patient's dementia from willful or perverse behavior. The story may elicit quite human reactions to senile behavior and evoke a discussion that is useful for developing self-knowledge and understanding required of a health care professional.
— id: 8167, year: 1998, vol: 73, page: 66, stat: Journal Article,

Medicine and the arts: commentary on "The Ship Pounding" and "The Sick Wife"
Aull F
1997 ;72(3):194-195, Academic medicine
— id: 19706, year: 1997, vol: 72, page: 194, stat: Journal Article,

The on-line database of literature, arts & medicine
Aull F
[New York] : New York University School of Medicine, 1996,
— id: 633, year: 1996, vol: , page: , stat: ,

The on-line database of literature & medicine
Aull F
[New York] : New York University School of Medicine, 1995,
— id: 632, year: 1995, vol: , page: , stat: ,

An on-line resource for the medical humanities
Aull F; Nachbar MS; Smith R; Brewer K
1994 Sep;69(9):737-738, Academic medicine
— id: 18943, year: 1994, vol: 69, page: 737, stat: Journal Article,

Specific drug sensitive transport pathways for chloride and potassium ions in steady-state Ehrlich mouse ascites tumor cells
Aull F
1982 Jun 28;688(3):740-746, Biochimica & biophysica acta
A major aim of this investigation was to determine whether, in steady-state ascites cells, Cl- transport can be partitioned into a furosemide-sensitive cotransport with K+ and a separate 4,4'-isothiocyanostilbene-2,2'-disulfonic acid (DIDS) sensitive self-exchange. Both Cl- and K+ fluxes were studied. The furosemide- and Cl- sensitive K+ fluxes were equivalent, both in normal ionic media and when the external K+ concentration, [K+]o, was varied from 4 to 30 mM. The stoichiometry of the furosemide-sensitive Cl- and K+ fluxes was 2 Cl-:1 K+ at 0.1 and 0.5 mM drug levels but increased to 3 Cl-:1 K+ at 1.0 mM furosemide. DIDS at 0.1 mM had no effect on the K+ exchange rate but inhibited Cl- exchange by 39% +/- 2 (S.E.). The effects of DIDS and 0.5 mM furosemide on Cl- transport were additive but 1.0 mM furosemide and DIDS had overlapping inhibitory actions. Thus furosemide acts on components of K+ and Cl- transport which are linked to each other, but the drug also inhibits an additional DIDS-sensitive Cl- pathway, when present at higher concentrations. The dependence of the furosemide-sensitive K+ and Cl- transport on [K+]o was also studied; both fluxes fell as the [K+]o increased. The latter results recall those in an earlier study by Hempling (Hempling, H.G. (1962) J. Cell. Comp. Physiol. 60, 181-198)
— id: 19700, year: 1982, vol: 688, page: 740, stat: Journal Article,

Potassium chloride cotransport in steady-state ascites tumor cells. Does bumetanide inhibit?
Aull F
1981 May 6;643(2):339-345, Biochimica & biophysica acta
Bumetanide is a potent diuretic drug which has some structural features in common with furosemide. The steady-state exchange of K+ and Cl- was investigated in Ehrlich ascites tumor cells treated with bumetanide. This agent did not alter the cellular content of K+ or Cl- but the self-exchange of both ions was depressed. K+ self-exchange was inhibited by 55% at bumetanide concentrations as low as 10(-6) M. Cl- self-exchange was less sensitive to this drug but at low concentrations (between 10(-6) and 10(-3) M) bumetanide was a more effective inhibitor of Cl- transfer than furosemide. The steady-state K+ flux of cells equilibrated in NO3- media was compared with the K+ flux in cells treated with 10(-4) or 10(-3) M bumetanide; the Cl(-)-sensitive K+ exchange was equivalent to the bumetanide-sensitive K+ exchange. Since the results suggested that a bumetanide-sensitive (Cl-, K+) cotransport could be operative in steady-state cells, the stoichiometry of the bumetanide-sensitive fluxes was determined by measuring Cl- and K+ fluxes simultaneously in the same cell suspension. At 5 . 10(-4) and 10(-3) M bumetanide concentrations, the ratio of these fluxes was 0.98 +/- 0.07 (S.E.) and 1.04 +/- 0.06, respectively, consistent with the postulated cotransport mechanism. At 10(-4) and 10(-5) M, however, the ratio of the bumetanide-sensitive Cl-/K+ flux was significantly less than 1.0. Since the magnitude of the bumetanide-sensitive K+ flux at 10(-4) M was close to that of the Cl(-)-sensitive flux, a ratio of less than 1.0 at this drug level indicates that Cl-sensitivity and drug sensitivity may not reflect inhibition of the same process under all circumstances
— id: 19701, year: 1981, vol: 643, page: 339, stat: Journal Article,

Inhibition of Ehrlich ascites cell anion transport by 1-isothiocyanate-4-benzenesulfonic acid
Aull F
1980 Jul;599(2):580-586, Biochimica & biophysica acta
The effects of 1-isothiocyanate-4-benzene sulfonic acid on steady state Cl- and SO24(4 transport in Ehrlich mouse ascites tumor cells were investigated. At 10 mM, 1-isothiocyanate-4-benzenesulfonic acid reduced SO24(-) exchange by 94% but Cl- exchange was reduced by only 37%; Cl- exchange was not further inhibited by as much as 60 min of preincubation with 1-isothiocyanate-4-benzenesulfonic acid. Inhibition of Cl- exchange was completely reversible following 30-45 min of contact with 1-isothiocyanate-4-benzenesulfonic acid whereas under the same conditions, inhibition of SO24(-) exchange was irreversible. The effect o 1-isothiocyanate-4-benzenesulfonic acid on SO24(-) transport could be reversed, however, when exposure to 1-isothiocyanate-4-benzene-sulfonic acid lasted for only 2 min. In these respects the action of 1-isothiocyanate-4-benzenesulfonic acid resembles that of 4-acetamido-4'-isothiocyanostilbene-2,2'-disulfonic acid and 4,4'-diisothiocyano-1,2-diphenylethane-2,2'-disulfonic acid; the results are compatible with separate membrane sites for Cl- and SO24(-) transport. The Ki for reversible inhibition of SO24(-) transport, determined from a Dixon plot, was 4.8 mM and the inhibition appeared to be non-competitive
— id: 19702, year: 1980, vol: 599, page: 580, stat: Journal Article,

Saturation behavior of ascites tumor cell chloride exchange in the presence of gluconate
Aull F
1979 Jul 5;554(2):538-540, Biochimica & biophysica acta
Steady state Cl- flux across the Ehrlich mouse ascites cell membrane was studied when gluconate replaced Cl- in the external medium. Saturation behavior was observed; K 1/2 was 23.9 mM Cl- and V was 758 micromol.g-1 dry weight.h-1. The cells lost K+, Cl- and H2O, consistent with relative impermeability to gluconate, and the Cl- efflux rate coefficient was elevated. The results indicate that a major portion of Cl- exchange occurs as a membrane transport process and suggest that the process is sensitive to intracellular Cl- levels
— id: 19703, year: 1979, vol: 554, page: 538, stat: Journal Article,

Chloride self exchange in Ehrlich ascites cells. Inhibition by furosemide and 4-acetamido-4'-isothiocyanostilbene-2,2'-disulfonic acid
Aull F; Nachbar MS; Oppenheim JD
1977 Dec 15;471(3):341-347, Biochimica & biophysica acta
The effects of furosemide and 4-acetamido-4'-isothiocyanostilbene-2,2'-disulfonic acid (SITS) on steady-state Cl- flux were studied in Ehrlich mouse ascites cells. At 10 mM, furosemide inhibited isotopically-determined Cl- flux by 86% without changing cell Cl- content, indicating that influx and efflux were depressed by the same amount. These results suggest that at least 86% of the steady-state Cl- flux may occur as a one for one exchange. Half of the inhibitory effect was not reversed by vigorous washing with albumin-Ringer. A smaller portion of steady-state Cl- flux was inhibited by SITS. The maximum effect of SITS was reached near 0.6 mM; at this concentration Cl- flux was reduced by 37% without an alteration in cell Cl- content. Possible competition of environment Cl- and SITS was investigated by replacing environment Cl- with acetate or NO3. These anions reduced the efficacy of SITS because they depressed cell Cl- turnover themselves, apparently acting on the same exchange process
— id: 18947, year: 1977, vol: 471, page: 341, stat: Journal Article,

Nature of lectin-induced alteration of potassium transfer in Ehrlich ascites tumor cells
Aull F; Nachbar MS; Oppenheim JD
1977 Jan;90(1):9-14, Journal of cellular physiology
The way in which the lectins concanavalin A (Con A) and Ricinus communis agglutinin (Ricin) alter the K+ content of Ehrlich ascites tumor cells was investigated. Unidrectional and net fluxes were determined in unwashed cells during a time course following lectin addition. Total influx, ouabain sensitive influx, Mg++- and Na+-K+-ATPase activity were all unaffected. Cell ATP content was normal for at least 19 minutes after exposure to Con A. Early after contact with Ricin or Con A efflux was stimulated 2-3-fold, resulting in net K+ loss, but after 20 minutes efflux had returned to normal. Ricin and Con A acted similarly although Ricin was present at only 1/50 the concentration of Con A. When the findings are evaluated together with previous work it is suggested that a particular membrane glycoprotein may be concerned in the efflux alteration observed
— id: 18951, year: 1977, vol: 90, page: 9, stat: Journal Article,

CHLORIDE EXCHANGE AND EFFECT OF SITS IN EHRLICH ASCITES TUMOR- CELLS
Aull, F; Nachbar, MS; Oppenheim, JD
1977 ;297(2):163-163, Journal of supramolecular structure
— id: 29576, year: 1977, vol: 297, page: 163, stat: Journal Article,

MECHANISM OF LECTIN INDUCED CHANGES IN POTASSIUM-TRANSPORT OF EHRLICH ASCITES TUMOR-CELLS
Aull, F; Nachbar, MS; Oppenheim, JD
1976 ;35(3):605-605, Federation Proceedings (Federation of American Societies for Experimental Biology)
— id: 29495, year: 1976, vol: 35, page: 605, stat: Journal Article,

Interactions of lectins with plasma membrane glycoproteins of the Ehrlich ascites carcinoma cell
Nachbar MS; Oppenheim JD; Aull F
1976 Feb 6;419(3):512-529, Biochimica & biophysica acta
Several aspects of the interaction of various lectins with the surface of Ehrlich ascites carcinoma cells are described. The order of agglutinating activity for various lectins is Ricinus communis greater than wheat germ greater than or equal to concanavalin A greater than or equal to soybean greater than Limulus polyphemus. No agglutination was noted for Ulex europaeus. Using 125I-labeled lectins it was determined that there are 1.6 and 7 times as many Ricinus communis lectin binding sites for concanavalin A and soybean lectins. Sodium deoxycholate-solubilized plasma membrane material was subjected to lectin affinity chromatography and sodium dodecyl sulfate-polyacrylamide gel electrophoresis. The lectin receptors of the plasma membrane appeared to be heterogeneous and some qualitative differences could be discerned among the electrophoretically analyzed material, which bound to and was specifically eluted from the various lectin affinity columns. The characteristics of elution of bound material from individual lectin columns indicated secondary hydrophobic interactions between concanavalin A or wheat germ agglutinin and their respective lectin receptor molecules
— id: 18952, year: 1976, vol: 419, page: 512, stat: Journal Article,

Concanavalin A-induced alterations in sodium and potassium content of Ehrlich ascites tumor cells
Aull F; Nachbar MS
1974 Apr;83(2):243-250, Journal of cellular physiology
— id: 18956, year: 1974, vol: 83, page: 243, stat: Journal Article,

Cell surface contributions to the malignant process
Nachbar MS; Oppenheim JD; Aull F
1974 Sep;268(3):122-138, American journal of the medical sciences
— id: 18954, year: 1974, vol: 268, page: 122, stat: Journal Article,

Purification of a hemagglutinin from Limulus polyphemus by affinity chromatography
Oppenheim JD; Nachbar MS; Salton MR; Aull F
1974 Jun 18;58(4):1127-1134, Biochemical & biophysical research communications
— id: 18955, year: 1974, vol: 58, page: 1127, stat: Journal Article,

The effect of external anions on steady-state chloride exchange across ascites tumour cells
Aull F
1972 Mar;221(3):755-771, Journal of physiology
— id: 19704, year: 1972, vol: 221, page: 755, stat: Journal Article,

Absorption of fluid from isolated intestine of the toadfish, Opsanus tau
Aull F
1966 Mar;17(3):867-870, Comparative biochemistry & physiology
— id: 19705, year: 1966, vol: 17, page: 867, stat: Journal Article,