Eddie Louie, M.D.

Trichomonas tenax as a possible cause of eosinophilic pneumonia and respiratory failure
Rahimian, J; Felner, K; Louie, E; Schwartz, D
2006 JUL ;23(7):335-336A, Infections in medicine
A 73-year-old man was hospitalized with dyspnea, fevers, chills, myalgia, diffuse pulmonary infiltrates, peripheral eosmophilia, and eosinophiha on bronchoalveolar lavage BAL). BAL also revealed the presence of Trichomonas tenax and Entamoeba gingivalis. The patient did not respond to broad-spectrum antibiotics but had a dramatic response to corticosteroids. The authors hypothesize that T tenax may have caused this patient's eosinophilic pneumonia and that it may be an unrecognized cause in other similar cases
— id: 66453, year: 2006, vol: 23, page: 335, stat: Journal Article,

Herpes simplex virus hepatitis: case report and review [see comments]
Kaufman B; Gandhi SA; Louie E; Rizzi R; Illei P
1997 Mar;24(3):334-338, Clinical infectious diseases
Hepatitis is an unusual manifestation of herpesvirus infection. Herpes simplex virus hepatitis is a difficult diagnosis to establish, and the infection is often fatal. We report one case of herpes simplex virus hepatitis and review 51 cases in the literature. Impaired immunity resulting from pregnancy, malignancy, immunosuppression, or inhalational anesthetics may be predisposing factors. Fever, nausea, vomiting, abdominal pain, leukopenia, thrombocytopenia, coagulopathy, and a marked rise in serum transaminase levels are invariably present. Liver biopsy is the procedure of choice for diagnosis. The liver appears mottled and has a minimal inflammatory response. Mortality rates associated with herpes simplex virus hepatitis are high, and early diagnosis and treatment with acyclovir or vidarabine may produce a favorable outcome
— id: 7177, year: 1997, vol: 24, page: 334, stat: Journal Article,

Subacute cerebellitis in Lyme disease
Neophytides A; Khan S; Louie E
1997 Nov-Dec;51(8):523-524, International journal of clinical practice
Cerebellitis is not a recognised manifestation of Lyme disease. We describe a patient with clinical features of subacute cerebellitis, cerebrospinal fluid (CSF) monocytic pleocytosis, positive CSF Borrelia burgdorferi antibodies, negative brain magnetic resonance imaging and a benign course after treatment with ceftriaxone. Possible earlier cases are discussed. Lyme disease should be considered in all cases of subacute cerebellitis
— id: 12141, year: 1997, vol: 51, page: 523, stat: Journal Article,

Severe polymyositis-like syndrome associated with zidovudine therapy of AIDS and ARC
Bessen, L J; Greene, J B; Louie, E; Seitzman, P; Weinberg, H
1988 Mar 17;318(11):708-708, New England journal of medicine
— id: 141807, year: 1988, vol: 318, page: 708, stat: Journal Article,

Treatment of cryptosporidiosis with oral bovine transfer factor
Louie E; Borkowsky W; Klesius PH; Haynes TB; Gordon S; Bonk S; Lawrence HS
1987 Sep;44(3):329-334, Clinical immunology & immunopathology
Cryptosporidia are intestinal protozoans long known to cause diarrhea in humans, especially those with acquired immune deficiency syndrome (AIDS). When transfer factor prepared from calves which possessed delayed-type hypersensitivity to Eimeria bovis was given to nonimmune calves and mice it conferred protection against clinical infection (coccidiosis). Recent studies with oral bovine transfer factor have shown that it can confer cell-mediated immunity to humans. Based on these findings we decided to treat eight AIDS patients suffering from Cryptosporidium-associated diarrhea with transfer factor prepared from calves immune to Cryptosporidium. Prior to treatment with transfer factor, three patients had been treated with spiramycin, one patient with alpha-difluoromethylornithine (DFMO), and one patient with furazolidone for greater than 1 month without clinical or laboratory improvement. Following administration of transfer factor, five or eight patients exhibited a decrease in the number of bowel movements and the development of formed stools. Cryptosporidium was eradicated from the stools of four patients but two of these patients subsequently relapsed and one patient continued to have diarrhea despite the absence of Cryptosporidium in the stool. One patient has been free of diarrhea and Cryptosporidium for 2 years after discontinuation of transfer factor therapy
— id: 14569, year: 1987, vol: 44, page: 329, stat: Journal Article,

TUBERCULOSIS IN NON-HAITIAN PATIENTS WITH ACQUIRED-IMMUNODEFICIENCY-SYNDROME
LOUIE, E; RICE, LB; HOLZMAN, RS
1986 OCT ;90(4):542-545, Chest
— id: 51148, year: 1986, vol: 90, page: 542, stat: Journal Article,

Cryptococcal disease in patients with the acquired immunodeficiency syndrome. Diagnostic features and outcome of treatment
Zuger A; Louie E; Holzman RS; Simberkoff MS; Rahal JJ
1986 Feb;104(2):234-240, Annals of internal medicine
Between 1 January 1981 and 1 December 1984, 34 of 396 patients with the acquired immunodeficiency syndrome (AIDS) developed cryptococcal infections. Twenty-six cases are reviewed. Twenty-two patients had brain or meningeal disease; the others had pulmonary disease (2 patients), pericarditis (1 patient), and antigenemia (1 patient). During treatment, 3 patients died of cryptococcosis and 3 died of other causes. Fifteen patients were followed for more than 6 weeks after treatment. Of 8 patients who received no additional amphotericin B, 4 had relapses and died of cryptococcosis within 6 months, 3 died of other causes, and 1 survived. Of 7 patients who received maintenance therapy with amphotericin B, none had relapses, 3 died of other causes, and 4 survived. Our data suggest that maintenance therapy with amphotericin may be needed to prevent relapse in patients with AIDS
— id: 38202, year: 1986, vol: 104, page: 234, stat: Journal Article,

Adrenal insufficiency as a complication of the acquired immunodeficiency syndrome
Greene LW; Cole W; Levy B; Louie E; Raphael B; Waitkevicz HJ; Blum M
1985 ;:289-291, Year book of endocrinology
— id: 36582, year: 1985, vol: , page: 289, stat: Journal Article,

Adrenal insufficiency as a complication of the acquired immunodeficiency syndrome
Greene LW; Cole W; Greene JB; Levy B; Louie E; Raphael B; Waitkevicz J; Blum M
1984 Oct;101(4):497-498, Annals of internal medicine
— id: 36564, year: 1984, vol: 101, page: 497, stat: Journal Article,